{"id":18021,"date":"2020-11-24T08:19:41","date_gmt":"2020-11-24T08:19:41","guid":{"rendered":"https:\/\/dermatly.com\/inhibidores-del-factor-de-necrosis-tumoral\/"},"modified":"2026-03-10T17:06:15","modified_gmt":"2026-03-10T17:06:15","slug":"tumor-necrosis-factor-inhibitors","status":"publish","type":"glossary","link":"https:\/\/dermatly.com\/en\/inhibidores-del-factor-de-necrosis-tumoral\/","title":{"rendered":"Tumor Necrosis Factor Inhibitors"},"content":{"rendered":"<section class=\"textBlock\">\n<h1>Anti-TNF Medications: Function, Mechanism, and Treatment of Chronic Inflammation<\/h1>\n<p>Tumor Necrosis Factor (<span class=\"term\" data-term-id=\"2290\">TNF<\/span>) is defined as a <span class=\"term\" data-term-id=\"433\">cytokine<\/span> essential directly involved in inflammatory processes <span class=\"term\" data-term-id=\"490\">and inflammatory<\/span> that affect multiple organ systems, including joints, skin, and the gastrointestinal tract. Understanding its molecular role is fundamental for developing effective treatment strategies against chronic diseases.<\/p>\n<p>The most advanced anti-TNF treatments are <span class=\"term\" data-term-id=\"466\">undetectable<\/span> <span class=\"term\" data-term-id=\"426\">Effective Diagnosis of Amyloidosis<\/span> designed to specifically neutralize <span class=\"term\" data-term-id=\"2291\">TNF-alpha<\/span> (TNF\u03b1). These cutting-edge biological agents are crucial for controlling systemic inflammation associated with conditions like arthritis <span class=\"term\" data-term-id=\"1854\">control<\/span> hyperpigmentation and hypopigmentation <span class=\"term\" data-term-id=\"148\">inflammation<\/span> systemic inflammation associated with conditions like arthritis <span class=\"term\" data-term-id=\"1578\">arthritis<\/span> and various dermatological inflammatory diseases. They pioneered the introduction of biologic therapies to the market.<\/p>\n<p>Among the main monoclonal antibodies that exert a direct neutralizing action on TNF\u03b1, the following marketed treatments stand out:<\/p>\n<ul>\n<li>Infliximab<\/li>\n<li>Adalimumab<\/li>\n<li>Etanercept<\/li>\n<li>Golimumab<\/li>\n<li>Certolizumab<\/li>\n<\/ul>\n<h2>Other Therapeutic Strategies with Anti-TNF Impact<\/h2>\n<p>Beyond specific monoclonal antibodies, there is a spectrum of medications that also modulate or exhibit a suppressive activity on TNF:<\/p>\n<ul>\n<li>Thalidomide<\/li>\n<li>Sulfasalazine<\/li>\n<li>Pentoxifylline<\/li>\n<li>Bupropion<\/li>\n<\/ul>\n<p>Additionally, research has pointed to the potential of compounds derived from natural sources to influence TNF modulation:<\/p>\n<ul>\n<li>Catechins<\/li>\n<li>Curcumin<\/li>\n<li>Cannabis<\/li>\n<li>Echinacea purpurea<\/li>\n<\/ul>\n<h2>Indications for Anti-TNF Drugs in Skin Pathologies<\/h2>\n<p>The use of anti-TNF monoclonal antibodies is fundamental in the management of chronic plaque psoriasis. <span class=\"term\" data-term-id=\"235\">plaque<\/span>.  Its application extends to those inflammatory skin diseases classified as severe, especially when conventional treatments have not offered the desired clinical response. These conditions include:<\/p>\n<ul>\n<li>Hidradenitis Suppurativa<\/li>\n<li>Hidradenitis suppurativa secondary to Crohn's disease<\/li>\n<li><span class=\"term\" data-term-id=\"1626\">Pyoderma<\/span> gangrenosum and the <span class=\"term\" data-term-id=\"350\">syndrome<\/span> PAPA syndrome (without specific branding)<\/li>\n<\/ul>\n<section class=\"textBlock imageLinkBlockTitle\">\n<h2 class=\"colour--primary\">Dermatological Conditions Treated with Tumor Necrosis Factor Inhibitors<\/h2>\n<\/section>\n<section class=\"imageLinkBlock\">\n<div class=\"flex\">\n<div class=\"imageLinkBlock__item__image\">\n                <img decoding=\"async\" src=\"https:\/\/dermatly.com\/wp-content\/uploads\/2020\/11\/severe-psoriasis__ProtectWyJQcm90ZWN0Il0_FocusFillWzI5NCwyMjIsIngiLDFd.JPG\" alt=\"Example of severe psoriasis on the skin, a condition treated with anti-TNF.\">\n            <\/div>\n<p class=\"p-small\">Severe psoriasis<\/p>\n<div class=\"imageLinkBlock__item__image\">\n                <img decoding=\"async\" src=\"https:\/\/dermatly.com\/wp-content\/uploads\/2020\/11\/hidradenitis-suppurativa__ProtectWyJQcm90ZWN0Il0_FocusFillWzI5NCwyMjIsInkiLDg1XQ.JPG\" alt=\"Illustrative image of hidradenitis suppurativa, another indication for anti-TNF therapies.\">\n            <\/div>\n<p class=\"p-small\">Hidradenitis Suppurativa<\/p>\n<div class=\"imageLinkBlock__item__image\">\n                <img decoding=\"async\" src=\"https:\/\/dermatly.com\/wp-content\/uploads\/2020\/11\/pyoderma-gangrenosum__ProtectWyJQcm90ZWN0Il0_FocusFillWzI5NCwyMjIsIngiLDBd.JPG\" alt=\"Illustration of pyoderma gangrenosum, a severe inflammatory disease treated with these drugs.\">\n            <\/div>\n<p class=\"p-small\">Pyoderma Gangrenosum<\/p>\n<\/p><\/div>\n<\/section>\n<div class=\"clearfix imageLinkBlock\"><\/div>\n<h2>Risks and Crucial Considerations for Anti-TNF Drugs<\/h2>\n<p>Generally, these anti-TNF biological agents are administered via subcutaneous injection. It is common to experience mild side effects localized at the injection site or reactions linked to the infusion stage. It is imperative to avoid prescribing these medications in patients presenting with decompensated heart failure or in advanced stages.<\/p>\n<p>The main risk associated with anti-TNF therapy lies in the immunosuppression it causes, which increases the patient's vulnerability to various <span class=\"term\" data-term-id=\"2101\">Unlike other<\/span>. This includes an increased risk of contracting infections of <span class=\"term\" data-term-id=\"937\">bacterial<\/span>, bacterial, fungal, and viral origin, highlighting serious pathogens such as:<\/p>\n<ul>\n<li>Tuberculosis<\/li>\n<li>Histoplasmosis<\/li>\n<li>Coccidioidomycosis<\/li>\n<\/ul>\n<p>Correct monitoring and the balance between inflammation control and infectious risk are fundamental pillars in managing patients under treatment with Tumor Necrosis Factor inhibitors.<\/p>\n<\/section>\n<ul>\n<li>Blastomycosis<\/li>\n<\/ul>\n<p>Furthermore, these treatments may increase the likelihood of developing certain types of skin cancer and worsening conditions of an <span class=\"term\" data-term-id=\"691\">defect<\/span>, autoimmune nature, such as demyelinating diseases.<\/p>\n<p>A documented clinical phenomenon is the potential decrease in the efficacy of anti-TNF medications over time. This loss of response is attributed, in part, to the induction <span class=\"term\" data-term-id=\"2099\">induction<\/span> of antibodies that attack the medication itself. Ironically, anti-TNFs can also trigger their own dermatological side effects, including:<\/p>\n<ul>\n<li><span class=\"term\" data-term-id=\"1475\">Pustular<\/span> <span class=\"term\" data-term-id=\"1130\">palmoplantar<\/span><\/li>\n<li>Various presentations of skin rashes.<\/li>\n<\/ul>\n<h2>Summary and Considerations Regarding Anti-TNF Treatments<\/h2>\n<p>In conclusion, anti-TNF drugs represent a substantial advance in addressing chronic inflammatory diseases, offering effective alternatives when conventional therapies fail. However, their use requires a rigorous evaluation of the risks associated with immunosuppression and the need for continuous long-term dermatological surveillance.<\/p>\n<section class=\"textBlock imageLinkBlockTitle\">\n<h2 class=\"colour--primary\">Dermatological Conditions Potentially Induced by Anti-TNF Inhibitors<\/h2>\n<\/section>\n<ul>\n<li>Psoriasis<\/li>\n<li><span class=\"term\" data-term-id=\"491\">Eczema<\/span><\/li>\n<li>dermatitis.<\/li>\n<li>Morphea<\/li>\n<li><span class=\"term\" data-term-id=\"824\">Diffuse<\/span> areata<\/li>\n<li>Vitiligo<\/li>\n<li><span class=\"term\" data-term-id=\"113\">Granuloma<\/span> annular<\/li>\n<li>Sarcoidosis<\/li>\n<li><span class=\"term\" data-term-id=\"90\">phenotypes<\/span> Erythema multiforme<\/li>\n<li><span class=\"term\" data-term-id=\"1091\">Vasculitis<\/span><\/li>\n<li>Stevens-Johnson syndrome \u2013 toxic epidermal <span class=\"term\" data-term-id=\"846\">epidermal<\/span> <span class=\"term\" data-term-id=\"1159\">shock<\/span><\/li>\n<li>Dermatitis <span class=\"term\" data-term-id=\"53\">Nummular<\/span> drug-induced<\/li>\n<\/ul>\n<section class=\"imageLinkBlock\">\n<div class=\"flex\">\n<figure class=\"imageLinkBlock__item__image\">\n            <img decoding=\"async\" src=\"https:\/\/dermatly.com\/wp-content\/uploads\/2020\/11\/hand-dermatitis-aTNF__ProtectWyJQcm90ZWN0Il0_FocusFillWzI5NCwyMjIsIngiLDFd.JPG\" alt=\"Image showing hand dermatitis as a potential side effect of anti-TNF treatments.\"><figcaption class=\"p-small\">Hand Dermatitis<\/figcaption><\/figure>\n<figure class=\"imageLinkBlock__item__image\">\n            <img decoding=\"async\" src=\"https:\/\/dermatly.com\/wp-content\/uploads\/2020\/11\/nummular-dermatitis-aTNF__ProtectWyJQcm90ZWN0Il0_FocusFillWzI5NCwyMjIsIngiLDFd.JPG\" alt=\"Example of nummular dermatitis observed in the context of tumor necrosis factor inhibitor therapy.\"><figcaption class=\"p-small\">Nummular eczema<\/figcaption><\/figure>\n<figure class=\"imageLinkBlock__item__image\">\n            <img decoding=\"async\" src=\"https:\/\/dermatly.com\/wp-content\/uploads\/2020\/11\/alopecia-areata-aTNF__ProtectWyJQcm90ZWN0Il0_FocusFillWzI5NCwyMjIsIngiLDFd.JPG\" alt=\"Manifestation of alopecia areata in a patient undergoing anti-TNF immunomodulatory treatment.\"><figcaption class=\"p-small\">Alopecia Areata<\/figcaption><\/figure>\n<\/p><\/div>\n<\/section>\n<div class=\"clearfix imageLinkBlock\"><\/div>\n<div class=\"grey-box\">\n<p><em><span style=\"font-size: 0.9em;\">For detailed information on these prescription medications, including their approved uses and risk profile, it is essential to consult the official data sheets approved in New Zealand, accessible through the Medsafe portal.<\/span><\/em><\/p>\n<\/div>","protected":false},"excerpt":{"rendered":"<p>Medicamentos Anti-TNF: Funci\u00f3n, Funcionamiento y Tratamiento de la Inflamaci\u00f3n Cr\u00f3nica El Factor de Necrosis Tumoral (TNF) se define como una citocina esencial implicada directamente en los procesos inflamatorios que afectan m\u00faltiples sistemas org\u00e1nicos, incluyendo articulaciones, piel y el tracto gastrointestinal. Entender su papel molecular es fundamental para desarrollar estrategias de tratamiento eficaces contra enfermedades cr\u00f3nicas. [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":14748,"parent":0,"template":"","glossary-cat":[],"class_list":["post-18021","glossary","type-glossary","status-publish","has-post-thumbnail","hentry"],"yoast_head":"<!-- This site is optimized with the Yoast SEO Premium plugin v26.2 (Yoast SEO v27.1.1) - https:\/\/yoast.com\/product\/yoast-seo-premium-wordpress\/ -->\n<title>Inhibidores del factor de necrosis tumoral - Dermatly.com - El sitio de tu piel<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/dermatly.com\/en\/tumor-necrosis-factor-inhibitors\/\" \/>\n<meta property=\"og:locale\" content=\"en_US\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Inhibidores del factor de necrosis tumoral\" \/>\n<meta property=\"og:description\" content=\"Medicamentos Anti-TNF: Funci\u00f3n, Funcionamiento y Tratamiento de la Inflamaci\u00f3n Cr\u00f3nica El Factor de Necrosis Tumoral (TNF) se define como una citocina esencial implicada directamente en los procesos inflamatorios que afectan m\u00faltiples sistemas org\u00e1nicos, incluyendo articulaciones, piel y el tracto gastrointestinal. 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